Philip Morris
Passive Smoking in New Zealand
Fields
- Author
- Reinken, J.
- Document File
- 2023511660/2023512308/Ets: Heart Disease 930900
- Area
- SCIENTIFIC AFFAIRS/BLACK LATERAL OLD S&T
- Type
- PSCI, PUBLICATION SCIENTIFIC
- BIBL, BIBLIOGRAPHY
- Master ID
- 2023511661/2307
Related Documents:- 2023511661-2307 Environmental Tobacco Smoke and Heart Disease
- 2023511710 the Relationship of Passive Smoking to Various Health Outcomes Among Seventh-Day Adventists in California.
- 2023511714-1718 Passive Smoking and the Risk of Heart Attack or Coronary Death
- 2023511722-1727 Effects of Passive Smoking on Ischemic Heart Disease Mortality of Nonsmokers A Prospective Study
- 2023511728 Erratum
- 2023511729 'effects of Passive Smoking on Ischemic Heart Disease Mortality of Nonsmokers: A Prospective Study'
- 2023511730 the First Author Replies
- 2023511734-1737
- 2023511738-1744 Passive Smoking in Females and Coronary Heart Disease
- 2023511749-1756 Original Contributions Heart Disease Mortality in Nonsmokers Living with Smokers
- 2023511760-1781 Lung Cancer in Japan: Effects of Nutrition and Passive Smoking
- 2023511785-1789 Passive Smoking and Cardiorespiratory Health in A General Population in the West of Scotland
- 2023511790 Passive Smoking and Cardiorespiratory Health in Scotland
- 2023511791-1792 Passive Smoking and Cardiorespiratory Health in Scotland
- 2023511793-1795 Passive Smoking and Cardiorespiratory Health in Scotland
- 2023511800-1802 Public Health Briefs Passive Smoking and 20-Year Cardiovascular Disease Mortality Among Nonsmoking Wives, Evans County, Georgia
- 2023511806-1816 Relationship of Passive Smoking to Risk of Lung Cancer and Other Smoking-Associated Diseases
- 2023511818 Increased Incidence of Heart Attacks in Nonsmoking Women Married to Smokers
- 2023511822-1824 Cvd Epidemiology Newsletter
- 2023511829-1841 Original Contributions Effects of Passive Smoking in the Multiple Risk Factor Intervention Trial
- 2023511842 Re: 'effects of Passive Smoking in the Multiple Risk Factor Intervention Trial'
- 2023511843-1844 Re: 'effects of Passive Smoking in the Multiple Risk Factor Intervention Trial'
- 2023511845 Re: 'effects of Passive Smoking in the Multiple Risk Factor Intervention Trial'
- 2023511846 the Authors Reply
- 2023511849-1853 Smoking As A Risk Factor for Cerebral Ischemia
- 2023511857-1862 Urinary Cotinine Measurement in Patients with Buerger's Disease - Effects of Active and Passive Smoking on the Disease Process
- 2023511865-1881 An Estimate of Adult Mortality in the United States From Passive Smoking
- 2023511882 Editorial Cardiovascular Risks of Environmental Tobacco Smoke
- 2023511883-1887 An Estimate of Adult Mortality in the United States From Passive Smoking: A Response
- 2023511888-1890 An Estimate of Adult Mortality in the United States From Passive Smoking: A Response
- 2023511891-1892 Ischemic Heart Disease: Response to Lee
- 2023511893-1895 Rebuttal to Lee / Katzenstein Commentary on Passive Smoking Risk
- 2023511896-1899 An Estimate of Adult Mortality in the United States From Passive Smoking: A Response
- 2023511900-1906 An Estimate of Adult Mortality in the United States From Passive Smoking: A Response to Criticism
- 2023511908-1911 Deaths From Lung Cancer and Ischemic Heart Disease Due to Passive Smoking in New Zealand
- 2023511912 Deaths From Lung Cancer and Ischemic Heart Disease Due to Passive Smoking in New Zealand
- 2023511913 Passive Smoking in New Zealand
- 2023511915 Passive Smoking in New Zealand
- 2023511916 Passive Smoking and Passive Thinking
- 2023511918-1937 Cardiovascular Diseases and the Work Environment A Critical Review of the Epidemiological Literature on Chemical Factors
- 2023511939-1950 Clinical Progress Series Passive Smoking and Heart Disease Epidemiology, Physiology, and Biochemistry
- 2023511952-1957 Review Passive Smoking and the Risk of Heart Disease
- 2023511958-1961 Aha Medical / Scientific Statement Position Statement Environmental Tobacco Smoke and Cardiovascular Disease A Position Paper From the Council on Cardiopulmonary and Critical Care, American Heart Association
- 2023511965-1983 the Health Consequences of Involuntary Smoking A Report of the Surgeon General
- 2023511985-1998 Environmental Tobacco Smoke Measuring Exposures and Assessing Health Effects
- 2023512000-2015 Environmental Tobacco Smoke Proceedings of the International Symposium at Mcgill University 890000 Environmental Tobacco Smoke and Cardiovascular Disease: A Critique of the Epidemiological Literature and Recommendations for Future Research
- 2023512016-2028 Panel Discussion on Cardiovascular Disease
- 2023512030-2037 Indoor Air Quality and Ventilation Environmental Tobacco Smoke (Ets) and Cardiovascular Disease
- 2023512039-2054 A Critique of the Methods Used to Assess the Toxic Effects on Man of Combustion Products.
- 2023512056-2066 Coronary Heart Disease and Involuntary Smoking
- 2023512068-2077 7. Environmental Tobacco Smoke and Coronary Heart Disease
- 2023512079-2088 Environmental Tobacco Smoke and Coronary Heart Disease
- 2023512090-2091 Editorial Give A Dog-End A Bad Name
- 2023512093-2108 Weaknesses in Recent Risk Assessments of Environmental Tobacco Smoke
- 2023512110-2129 Environmental Tobacco Smoke and Mortality A Detailed Review of Epidemiological Evidence Relating Environmental Tobacco Smoke to the Risk of Cancer, Heart Disease and Other Causes of Death in Adults Who Have Never Smoked - 5 Heart Disease
- 2023512131-2155 Environmental Tobacco Smoke Exposure and Occupational Heart Disease
- 2023512157-2171 Passive Smoking and Coronary Artery Disease. Biological Plausibility and Severity of Effect
- 2023512173-2180 Carbon Monoxide and Cardiovascular Disease: An Analysis of the Weight of Evidence
- 2023512185-2189 the Effects of Passive Inhalation of Cigarette Smoke on Excercise Performance
- 2023512192-2195 Effect of Passive Smoking on Angina Pectoris
- 2023512199-2202
- 2023512203-2213 Effect of 'passive' Smoking on the Physical Load Tolerance of Coronary Heart Disease Patients
- 2023512216-2220 Indoor Passive Smoking: Its Effect on Cardiac Performance
- 2023512223-2224 Passive Smoking Severely Decreases Platelet Sensitivity to Antiaggregatory Prostaglandins
- 2023512227-2230 Platelet Sensitivity to Prostacyclin in Smokers and Non-Smokers
- 2023512233-2237 Besitzen Passivraucher Ein Erhohtes Thromboserisiko?
- 2023512241-2244 Passive Smoking Affects Endothelium and Platelets
- 2023512247-2253 Lipoprotein and Oxygen Transport Alterations in Passive Smoking Preadolescent Children the Mcv Twin Study
- 2023512256-2257 Abstracts of the 30th Annual Conference on Cardiovascular Disease Epidemiology Children's Hdl-Chol: the Effects of Tobacco: Smoking, Smokeless and Parental Smoking
- 2023512261-2266 Passive Smoking Alters Lipid Profiles in Adolescents
- 2023512269-2274 Serum Lipids & Lipoprotein Profiles of Cigarette Smokers & Passive Smokers
- 2023512278-2279 8th Worldconference on Tobacco or Health Building A Tobacco-Free World 920330 - 920403 Buenos Aires - Argentina Abstracts, Posters and Videos. Serum Lipoproteins in Nonsmokers Chronically Exposed to Tobacco Smoke in the Workplace
- 2023512282 the Association Between Carotid Arterial Wall Thickness and Active and Passive Cigarette Smoking
- 2023512285 Passive Smoking and Carotid Artery Wall Thickness: the Aric Study
- 2023512290-2297 Passive Smoking Increases Experimental Atherosclerosis in Cholesterol-Fed Rabbits
- 2023512300-2301 Supplement to Circulation Abstracts From the 65th Scientific Sessions New Orleans Convention Center New Orleans, Louisiana 921116 - 921119
- 2023512304-2307 Association of Passive Smoking with Increased Coronary Heart Disease Risk Is Not Explained by Elevation of Leucocyte Count
- Characteristic
- EXTR, EXTRA
- MARG, MARGINALIA
- Named Person
- Helsing
- Hirayama
- Kawachi
- Lee
- Hirayama
- Litigation
- Okag/Privilege Withdrawn
- Okag/Produced
- Author (Organization)
- Ffms Consultants
- New Zealand Medical Journal
- Site
- R529
- Date Loaded
- 24 May 1999
- UCSF Legacy ID
- eic02a00
Document Images
27 SEP'i'EMBER 1989 NEW ZEALAND MEDICAL JOURNAL /v'2, ( SW '1 6)'
r~ Passive smoking in Mew Zealand
Mr Lee's letter )11 pretends to a scientific basis it does not have.
Hirayama's first publication 12) i focused~ on cancer of the lung
among nonsmoking J apanese wives and set off a flurry of criticism
of the methodology-including the analysis. The analyses have
been redone showing significance enhanced'by improved analysis.
,nly someone committed to nonsense would report a p-value for
!he difference between the two results..
Mr Lee's criticism of the study by Helsing et al 1s)'makes no
sense on the face of~ it. Controlling for 'a whole range:of possibly
relevant confounding factors' has as much likelihood of
heightening the significance as of lowering it'. The researchers
found that adjusting the relative risks has in fact enhanced the
significance of their findings.
Mr Lee has a greater tolerance for assessing a study as
published' than most'scientists do,,as demonstrated by his tenth
reference. Perhaps he gives more weight to studies of 9 subjects
which unsurprisingly fail to yield significant results than most
epidemiologists would. He may not, however, show so little
tolerance for the epidemiological methods he exploits. Spousal
smoking has, again and againbeen shown to be associated with
lung cancer risk 14.5s). The biomedical underpinning-proven
studies in animals and dose-related responses ih hn*nAna_relating
the constituents of both sidestream and mainstream tobacco
smoke to production of cancers of: the lung is undisputed (7.81-
Propinquity of the nonsmoker to the smoker over time rather
than the concentration of single toxic substances in the ambient
air determines the degree of exposure. Given the large numbers
of exposed nonsmokers even a very low degree of risk has
substantial impact.
Misclassification bias, a favourite theme of Mr Lee, is a two-
edged criticism. As long as misreporting of exposure is as likely
for cases as for controls misclassification depresses the relative
risk. The risk will be overestimated only when cases whose
husbands smoke denyy their own, actual smoking more readily than
cases whose husbands do not smoke or when cases exaggeratee
their husbands' smoking more than controls do. Where actual
exposure has been measured and compared with reported
exposure the agreement has been high and the misreported
exposure has not been in only one direction.
The validity of extrapolating exposure in the home to exposure
at work raises other questions about indoor air. If the home setting
is one where a nonsmoker can choose another room to be in than~
the one the smoker is in, then exposures at home would be lower
than ~worksite exposures. In the workplace freedom to move away
from the smoke source is generally denied. By extrapolating
Kawachi et al )9) have probably underestimated the risk and the
number of deaths attributable to passive smoking.
Common sense does more than pseudo-science can to produce
credibility. The weight of the evidence is against Mr Lee and
others whom the tobacco interests sponsor )tol.
J Reinken, FFMS Consultants,
W ellington.
1. Lee PN. Deaths from lung cancer and iecheemic lieart'disease due to passive.amoking
in Ne.° Zealand. NZMed J 1989'; 102: 448..
2. Hiuayams T. Nonsmoking wivea of heavys smokers have a highernsh of,lung n.nae-
a study fromJapan..Br Med J 1981; .282a 183-5'..
3: HhJaing KJ. Sandler DP: Comstocjf GK'. Ch'eeE. Heartt disrise.fn, nonsmokers living
.itM smokers. Am J Epidemiol 1988; 127: 915-22.
4. Abelin T. Curreno t.rends in the epidemiology of smoking:.p..sive smoic'utg and lung
uncer. Sch,veie Rundach Med Prs: 1989; 76:: 87-92.
5.. Svendsen.KH. Kuller LH. Martin MJ. OcYene JK. Effects of passivesmohing:in the
multiple risk factor intervention trial. AmJ EpidemioL 1987:.126: 783-95'.
6.. Svendsen.KH. Kuller LH.. Re: 'Effects of passive smoiung in rth'e multiple ruJc factor
intervmtion trial'. AmJ Ep,idemiol19895 129. 226-7.
7:. US Departmmt of Health and Human Servioee:'rAe health oonsequeaaes of invohSntary mdcing" a
report of.the Surgeon-Cieneral. US DH1iS.~ Washmgwn..1:986.
8.Saraca R:.Paasivesmolcing and lungcancen. In: Zaridse DG: Peeo R. ads. Tobaoco:, a
major international health hazard..lnternationd Agency, for Research on Cancer
Scientific Publications No 74:.lARC..Lyon; 1986..
9. K.vachi 1. Peartc NE. JarYsm.RT. I1vtls from lungcanm~ and iscti'emic heart diseasr
due to passive smoking in Nev.Zealand'NZMed J 1989, 102. 337-40.
10. Martin P. Pa.sive smoking. NZ Med J 1987:,100: 69&7.
Cancer registration working group
We regret that Dr Hitchcock (NZ Med J 1989;,102: 419) regards.
our letter on cancer registration )1] as incorrect. We can only
repeat what'actually occurred.
Dr Hitchcock mentions a submission from the Board of Healt'h.,
515
After two letters from the group seeking details the board finally
stated:
'In reply to your letter of 22 October 1987 we believe there
is nothing to be gained from pursuing,the matters you raise
in your letter. Our reference in our original letter referred
to apparent breaches in the past and the need to provide
effective controls.'
That is as much information on, 'instances of breaches of
confidentiality''as was ever received from the Board of Health
despite the repeated requests from the group for information on
actual instances. The board did not refer to any submission from
private pathologists.
We repeat `no individual, no doctor and no group provided the
working group with information on breaches of~confidentiality''
Ill. The essential point' is that, despite all our efforts we could
not find any substantiated evidence of an actual breach of
confidentiality by the New Zealand Cancer Registry.
It should not be necessary, but it may be helpful, to emphasise
that had the group been given information on~ any instance
apparently involving a material breach of confidentiality we would
have regarded this as a serious matter and sought to ensure a
thorough, , independent and' sensitive investigation.
We would like to take this opportunity to thank the many
organisations and ~ individuals who submitted comments on our
report to the ReviewCommittee on Health Statistics. We
appreciate the constructive criticisms and the general support for
our proposals.
K R Cooke,, Department of Preventive and
Social Medicine,
University of Otago Medical School;
Dunedin;
A J Gray, Cancer Society of New Zealand;
W ellington;
A F Burry, Department of Pathology;
Christchurch Hospital,
Christchurch;
R Stewart, Department of Surgery,
Wellington School of Medicine,
Wellington.
1i Cooke KR. Gray AJBurry. AF. Sce.vart RJ.. NZ MedJ 1989; 102::197.
Dietetic advice
I was interested to ~ read the paper Children's diets:, what do
parents ad&and avoid? by Dr R P'K Ford and colleague (NZ Med
J 1989;, 102: 44'3h, with the analysis of advice on various food
substances.
It is quite staggering to find that none of the 103' children
interviewed for this article had been given dietetic advice. Over
and over again we are concerned to find that general practitioners
give detailed advice when ~they are not trained to d'o so. The whole
question of diet and nutrition is underestimatedand'undervalued
in the undergraduate and postgraduate curriculum,
Fortunately we have an efficient training programme for
dietitians in New Zealand and, in my opinion, it is unethical and
unprofessional ito attempt to give patients detailed advice on diet
when we have well trained and qualified colleagues available to
undertake this task.
I was provoked to write such a letter because all too frequently
we have people referred to hospital with complications of'diabetes
who have never had the opportunity to have'a consultation withh
a dietitian, who could certainly have influenced their eating
patterns.
D W Beaven, Department of Medicine,.
Christchurch Medical School;
Christchurch,
Informed consent
I recently received a copy of the New Zealand Medical
Association's revamped~ informed consent/request for treatment
form..
It is impossible for a patient to know that helshe has received
an adequate explanation of risks etc when the patient is in no
position to assess this. If any aspect of the operation is withheld
or overlooked : the patient has no way of knowing.
