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. ~~ 1- t ~ i ~!~>l~~,+1~l~#>~~1~'141I~'fl3*Tli4 (r±r). TC LDL HDL LDL,'HDL (mmoi.'L) (torool,I) (mmol/L) apoA1. (Z'L) apo8(j/L) apoD/tpoAJ ' 9'f !1 1@ - 26 4.1+-_o.6a 2.31t_n.6a 1.41_0.16 1.1e!:o.5a 1.27to,24 O.71t0.17 0.11lto.19 + 20 C.li_7.61 2.s2to.se T.29c0.16• 1.96t'0.60 1.11to.23• 0.74t0.1-4 037,±Ot22 tat~Ct~ - 9 5,15=?.66 3.35=0.i6 1.26_0.21 2.t5±o.79 0.95to.16 1.03t0.17 1.21rt0.10 + 24 l 5.6oce.13• 3.:5_0.T1 1.12_0.17` 3.42±0.74' 0s61to.13• 1.16t0.20 1.36t0.22' 3~ 3 s~i•;~t',P'F~'~;~ffJs9t1¢ Lo~isticle79~~f~~ AA2f Bi ror(J9i) s;(ai) STD(Bi) OR 09 !+ It 411A A I J. 0.406 0.069 0.013 1.611 1.500 4 16.93 <0:01 &EiLiliifall:ts, 0.714 0.05z 0.227 3.147 2.042 9 9.90 <O:oi 't; X~'x kt f• IiF N r' SJ ~6 9c7~f~eOt.~'.7~~T~i, ^J Ce.7~.tta7L-.`.~9a 1L1l7ldi Scot.tTA o,. VJ. BJYaJ7Y1 X7CE 7F C7 iIJ1/6 2 X , iff anJt>fia 9 ~t I & M 9 9~;K it tb:g a~': ~~1~~EFt~r~ COHb 7k'F 89 M 3+?w. Z9•)~'sG`a7t t M 3 : 11. Aromow Rt 10 $~~t~~~ R-11i A aA,IStE ARTO ~`•J121; '$'. 2'iJ.Bf F~ R COHb 3i-7% I Mt, * j)f4 r iA'K.L~9'•7Bt A ,E 1,'3: #LM ;1~ 4 t2 Bt3. a a ®', 1 33.88"or Mt;kik 6100"). t~+Jtl~ a 'B'*R'M ~iR'c. LfTi'j%r: ~It rA a r3i -4. ±;a I. Women's Passive Smokin{ and Coronary Heart Disease He Foo, ef ol.. Departrnent of Fpide- miology FonrtA ltilitar. .11e4iral Coliego of PLA. Xi'an Thirty-four, women CHD cases (7_ casrs d,aEnosed by coronary arverlojraphy and 12 myocardial i°farctuon) and 66 oh non-CHD coatrols (34 hospiul based and 34 populatuon based), matched on age (within five years). race. residence. occupation (and casc is to control as 1:2), were interviered re=ardinlt the smoking habits of themselves and their hnsbands The odds ratio (i. a. : OR) of .o°-smokin6 women CHD associated with I+aviag a smoking husband are !.[0+3.52, OR /SfGC/ do not include 1. Signifieaat dose-response relationship. betsveen OR of snome°'s CHD nd their husband's eiaarerte consumption. deratioa of passive smok'ih6 and eumulative quantity ot passive smoking were found ia the study. The logistic regression model anal'ysis rithh othen CHD risk' factors aho.ed that the relktionsbip with CHD and passive smok'ih6 still eaiated'. 11, was found that the metabolism of HDL-ebolksterol and apolipoproteins with passive smokers was abnormsl. 1. Scott TW. es al'. ($Ef;{i#). ~7)~lA7{9r18A : IM. Z~S6d41T'~K ' 198411 3(2):30.. 2. Stanton A.(k7 Ri#).i52flk®J~(dI~)~(!$l6~Ac ,Ts07a16'aft(f'i1.. ®4h~C#-Itg~c#Slll11966, 3:226. 3. Kaufi.mann F, et al. Adult passive smokinj in rhe home e°.iroome°tr-a risk factor for ehroni. airflosrw lim:ut.ioa.Am 1 Epidemiol 1963, 1117, 269. 4. Trschpoulba D. et all Lung cancer and passive smoking 1'nn J Cancer 1961, 29 1. 5. Aronor WS Effect of pass„•e smoking on a°ginl pecroris. N En61' 1' Med 1976: :99:21. 6. Garland C. ct at'Effects of passive smoking on ischem,c heart disease mortaliiv of nonsmolera Am 1 Ep,dem,ol 19951 121,645,

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