Tobacco Documents Online

0 0 ;~'5 THE NEW 1rGFd: ~} foreign matter onthe tlningg of el NEI~ CLUE FOU~(D ~e respiratary traci .s:~' w7 ~~ New York, Nt rk The Calit°rnia scientLsts su g December 9 `1959 ~~FOR LUNC CANCER gest that impairment of this , function byy smoke and oiga• . permit' the" moking In Cancer ~~ t:- CHICAGO (AP) - A man who x+ , Z2 bas: devoted• his scientific career -"toa studyofthe causes of-cancer ---• contended, Saturdaythat, air poll ~ ~ lution ~~ is a more important fac tor than cigarette smoking in the iocrease in lung cancer: r " a{ " Dr. Wilhelm C. Hueper, chief . of the Environmental Cancer ( Section. National Cancer Insti'- tute, Bethesda, Rld., said' an up- : surge in lung cancer first was ~.-- Doted between 1900 and 1920,, sev- ela6 years before th.e~practice~ot cigarette smoking became wide- apread. .,... •. _. , : ~ And he added that exposure to cancer-causing agents must occur ,; `~.!~.'at.least. 30~to:15 yeazs betore an increase in such deaths is de- ,v' 4-•.:tected statistically. Hueper made his remarks dur 'Ing an interview at the annual meeting of the American Assn. fon the Advancement of Science ?'.where he received the $031) :-' AAAS-Anne Franco Rosenthal ~. : bfemoriali Award for cancer, re• .-search. =ir <, 1iTi.; _ Hueper, 6a, said exhaust fumes ' from gasoline and coal tar by- products spewed into the atmos- :~phere b:yy industry are the princi- pall agents responsible for the sharp increase in lungg cancer• Howevet, he said smoking may play a role. He said tobacco -noke eontains small concentra- tions of cancer causing agents and that.smoRealso: can aot'as ;: an irritant that't weakens the lungs' normal' defense mecha- nism. ~ v.7 ~4 lX: ore Deadly Than " " _osa smoke may~ rettee ~~, abnormalretention: of cancer-~ ,}~z 4 q r.~~ ~i G. ... ... ..., . causing chemicals there "' RY,. :Ctgarette Smoke and Smog x - d' rog Esophagua Lse ~' ~e Said to Impair Respiratory • ~EviAene'e tlut variouets s aga; ° ,_ ean affect ciliary acttvit Tract Defense Ability y and~ . °~;4yrn Y -mucous ftowhas 6oen accumu+ ...,•.lating for several years. The_-~ Clgarette smoke snd afr po1• new study was contined to the ~'lutants In smog mayy contribute effecSs produeed, byy smog and~ ~okefromcigattttes- ~t~• ,bo.the.develapmentorlunB can- The scientists used the c111+ . 4~eee by fnterfertngwfth the ated lining of the esophagus flluds and minute hair-like and adjoiningportions of the °sttuctures ihthe Ilhing of the ~testlnal tract of frogs~asrep• resenta[ive ofsimllar tlssue tn- ~.lesptratory tract, -•. ,,•-: ~. Thls ossibfllt . was suggest- the respuratorytract 1n~ mam• p y mals such as man- Parallel ed In two reports from sclen• atudles on mammals permitted atstsat the. Unlversityof the relation of their findings . "Southern California school of on frogs to higher aniinmals, they -. Medicine that appear In the Wrote. rx•{i ' November number of The Jour- Smog and clgarette smoke ~ r~'M1 =~IW of the National Cancer In• tissue ~a btbrm~ °vn °r agairut the,., ` ue tn plastle charnbers- The ~. . , ._ stitute, just outl action of the mucous and cilia ':. . According to the reports,+nat, were measured after two-second lt~~.urali and, artificial smog and exposures to theirritants• ~~.thelr components as weili as The first~ reaction was an In+Ja .,•; ~' amokee from both filtered and ereased flow of mucous. This '. nonfiltered, cigarettes,, affect was followed Immediately, the '!thito tt'defeiti ~• ..e respraryracsens scensts wrote, by a sudden mechanisms In three ways. .. drop In theYlow, then~a gradual They. slow the flow of the return to normal functlon- -;~-, ' mucous stream, change the physkcali properties of the mu- 'eous and decrease the w~hiplike atUon~ of' the fine structures ialled cilta. Normal functioning of the mucous andciba are known to • preventt the secumulatton of He told newsmen he quit smoking 30 years ago. - .- ~ Pollution from car exhausts fumes could'~ be halled, he said4 . jf auto makers could' devise an engine that' causes complete com- btistion oL gasoline. Industiial' plants, he went on, should'~seek to •pertecti a system ~ whereby the air workers breath would be free [romvapors, gases and fumes•.. ~ NEW YORK TB*> S SMOKING TEST NEGATIVE New York, New York ^-- ",a Cholaterol Lev 1 BI d e s in oo ;• Nove'ltber 1'1+, 1559 Aro- Reported' Unchanged - •=-CHICAGO, Nov. 13(UPI)- , Smoking appears to have no .. ~•etf ~t on blood cholesterot lev- ~els, Dr„ Irvfhe t~ Page Cleve- rand~'hearL speclaliit, repurted today. He satd thathe and twoas- , sociates had eoneluded after a study that tf smoking played a role In causing heart attacks 1tt wasnot through any effect on cho]esterol. , Cholesterol is a fat-like sub- 1 Stance ln the blood..Ithas been ^"~+~ Impllcatedas a ea h l .Kn useor eart • ~~) attacks.\ianylnvestigatonbe- ~Ieve there lsevtdence of a con- O nectlon between hlkh cholesterol O leveisand hardening of the 'arteries. The reportt by Dr. Page was ~. ln TiLe Journal of the American btedi:caZ'hx~eclaLypp,. The second phase of the re- sponse. In which mucouss flowdecreasedand the defense mechanism ~ was presumably.iin- p~airedwas observed even at minute concentrations of it•ri, tants front smog and cigarette smoke. U.S. NE:1S & WORLD REPORTS Washington,, D. C. November 30, 1959 to have no effect on choliesterol levels in IJt.US`iti~..,u3C SMOKING. Smoking appears blood, according to a study published in the "Journal of-the American Medical Association." Dr. Irving H. Page, Cleveland heart speciali'~st, and two associates report that, if smoking plays any role in heart attacks, it is not through ~fi• aholesterol--the fatlike substance that has been linked with heart disease.