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WDRASl1OP: CARBON MONOXIDE AAI) CVD I Most potentially hazardous substances could be-controlled by these proce-- dures, thus attaining goals industry ideally should seek. ACKNOWLEDGMENTS Gnteful apprecialion is F'tcn to Eric P. KinduaJl. 11.D., and Lillian S. Nelson. ht.D., whose knorlcdFe and e•alualions aided in prepzration of this repon. Thanks arc given to Mrs. 1d. Wos. Lbnrian, Sl. Luke's Hos,^i:al of 51l..aukce. and her staR for assistance. . REFERENCES 1. Adams. 1. D.. EricY.sen. H. H., and Stone. H. L- Vlocardial mclaholism during exposure to carbon mono.ide in the conscious dog. J. Appl. Plrc.inl. 34, ?38-242 (1971 2. Anderson, E. N'., Avdclman. R. J.. Sirzuch. 1. hl.. Fonuin. N. 1.. and Kndson, J. H. Eftect of lo-le~el carbon monnxide exposure on onset and duration of angina pectoris. Ann. Jnrrru. .llyd. 79, 46-50 (1973). - - 3. Anderson. R. F.. Allcns,oah, D. C., and DeGrool, \S'. J. ,\l,ocardial toxicity from carbon monoxide poisonicp-Ann. Lurrn. Med. 67, 1172-I I6' (1967). 4. Aronow. V1. S. Effect of carbon monoxide on angina. intermittent claudicalion- and hemodynamics.ln "Clinical Implicalions of Air Pollution Rese_arch," pp- 107-1 l l. Publishing Sc. Group. Littlaon. hizss.. 1976. . 5. Arono.c. N'. S. ERen of cirarette smoking and of carbon monoxide on coronary hcan disease. Chest 70, S14-SIF t0.tober. 19761. 6. Aronoa. H'. S., Go!dsmith. 1. R.. Kern. 1. C.. Cassidy. J.. Felson, H'. H.. Johnson. L. L, and Adams. N'. Effect of :noking ciFaienes on cardiocascular hemodpnamics. Arch. En,lrnn. Hra6h 28, 330-33_' I I9-41. 7. Aronor.'. N'. S.. and Is40. V. H'. Carbon monoxide effect on exercise-induced angina pcaoris. Ann. Jnirrn. .Sfrd.79, 39_-39t (1973) 8. Astrup.P.ERecoofhspouzandofcarbonmonoxidcaxposuresonexperimemalaiherorclermis. Arm. Lrrcrn. Afrd. 71. C6-4.7 (1969). - 9. Astrup. P. Pathological effects of moderate carbon monoxide exposure. Snnrh 32, 20-31 11972) 10. Astft. S. F1.. Afiller. H. S.. Gregory, J. J.. Gianelli. S.. Jr., and Penny, L. J. S}sicrnic and mo'ocardial hemodtnunic responses to re]atiselg small concentralion of_ earboi)hemnglobin ICOHbI. Arrh. Enrirnc. Hrn!rh 18, 639 (1969)- IOa. Ball. E- G_ Sirittm:.lter. C. F.. and Cooper. O. The reaction of carbon monoxide. J. Binl. Chrm. 193. 635-647 (19e1). - 11. Blackburn. H. Electroczrdiegraphic classification for population comparisons: The hticnesota eode. J. Elcrrrorcrd:n!. 2. 3( 1969/. - - I Ia. Chance. B.. Erecrosha. V.. and %s'agner, hf. Mitochondrial responses to CO toxicitl. dun. A'.)-. Arad- Sri. 174. 19i-.04 (19701. _ 12. Conn. B. C.. Black, M. 1.- and ,fcHenry, P. L. Echocardiographic findings after acute carbon monoxide poisonince Brn. Hcnrr l. 38, 7L'-717 (197,61. 13. Dzcin. D. N. The app'icztion of LAreshold limit values for carbon monoxide under condirinns of continuous exposure. Acn. OcnrP- H?'F. 18, 21 -28. (1975). N. _E}asias, R. F., Topping. D- L., and Turner, D. M L The effect of inlcrmittenl carbon monoxide exposure on expcru=entel atherosclerosis in the rabbit. Arherosrlerosit 24, 5-'7-536 09761. 15. Eckhardt. R. E., KacFriznd. H. N.. Alarie, Y. C. E.. and Busey. A'. hf. The biologic effect for . long-ICrm exposure o.rpri:nztcs to carbon monoxide. Arch. £nriron. Health 25, 381-387 (1972) 1Sa. Goldbaum, L. R.. Ramierxz. R. G.. and Absalon, K. B. Rllat is the mechanism of carbon monocide toxicit- %? Aricrion Space EncLon. .Ved. 46, 1289-1'_91 (Oct. 1975). 16. Goldsmith, J. R., and .4mnow. W. S Carbon monoxide and coronary heart disease: A resicw-. Enrlror.. Res. 10,'_?6-:.'S (1975). 17. Hemberp, S. The Finnish foundry project-Background and general melhodology. Srnnd. l. ltbri En.iron- HeeAF. 2, suppl. I. 8-12 (1976). _ 18. Hemberg. S., Kar3.5. R-. Koskela. R-S., and Luoma. K. Angina pecloris, EC6 findings and blood pressure of foundn- vorkers in relation to carbon monoxide exposure. Srmre- l. tl',vA Enriron. Health 2, 54-63 (1976)- -1 1`~p ^ t` 'i1 L ~ - . .I I ~ l