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Lorillard

Lung Cancer, Coronary Heart Disease and Smoking

Date: 10 Mar 1982
Length: 12 pages
03607681-03607692
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Author
Evsenck, H.J.
Alias
03607681/03607692
Type
REPT, OTHER REPORT
Area
LEGAL DEPT FILE ROOM
Site
N14
Named Organization
Bethlem Royal Hospital
Maudsley Royal Hospital
Named Person
Bernard, C.
Evsenck, H.J.
Fisher, R.
Kissen
Spielberger
Tarrant
Woolf
Date Loaded
07 Jan 1999
Master ID
03607523/8364
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Author (Organization)
Univ of London
Litigation
Ppla/Produced
Characteristic
EXTR, EXTRA
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UCSF Legacy ID
pjv99d00

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Page 11: pjv99d00 Log in for more options!
165 controlled for in studies of the effects of smoking; inhalers do not on the whole differ from non-inhalers in disease proneness; the statistical relation between cigarette smoking and CHD disappears in many countries, e.g. Finland, Holland, Yugoslavia, Italy, Greece and Japan; there is an absence of dose-response relationship, i.e. there is little or no relation between duration of heavy cigarette smoking and risk of myocardial infarction; and the correlation between number of cigarettes smoked and CHD is not linear; ex-smokers in some studies appear to be safer than non-smokers; some types of CHD, such as angina pectoris (which comprises some 20;% of CHD in men) fail to show even a statistical correlation with cigarette smoking; some types of smoking (cigar, pipe) fail to show even a statistical correlation with CHD; etc. These are anomalies or failures of the causal theory which demand an explanation before the causal theory can be accepted. Some of these facts are much more readily explained in terms of a genetic-personality theory; thus the differential effects of cigarette vs. pipe/cigar smoking may find an explanation in terms of the known differences in personality type associated with these different smoking patterns. The general conclusion would seem to be that in the• case of CHD, as in the case of lung cancer, proof for the causal influence of smoking is still lacking and is by no -10-
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What is certain is that at the moment no final decision be made about whether or the degree to which cigarette 169 STATEMENT OF DR. PAUL I am Paul Miniard. I have e from the University of Florida, and si been Assistant Professor of Marketing University. My specialty is the study and I have published articles in that such journals as the Journal of Market of Consumer Research, and the Journal For over two years, I have w Blackwell, Professor of Marketing at t and author of, among other things, Con widely adopted textbook in the field. in reviewing and analyzing the cigaret tising provisions of S. 1929, the comp H.R. 5653, and the FTC Staff Report on Investigation. On Friday, March 12, D before the House Subcommittee on Healt provide his analysis of H.R. 5653. He mony concerning S. 1929, but because o sional commitment is unable to be here me to present the attached statement. can smoking may cause lung cancer or coronary heart disease, how it interacts with other factors (stress; personality), or how we can best protect the health of our citizens in relation to these diseases. "In ignorance, abstain!" warned the famous French scientist, Claude Bernard; hasty action on the basis of partial knowledge is unlikely to be in the best interests of those most concerned, namely the prospective victims of lung cancer and coronary heart disease. HANS J. EYSENCK -13-

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